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Description
Supporting the notion that TERT and TERC deficiency might contribute to pulmonary fibrosis by mechanisms dependent on telomerase activity but not necessarily telomere length, telomerase activity is induced in IPF and NSIP fibroblasts and systemic sclerosis lung compared to healthy donor samples (Fridlender et al., 2007
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pRB1 phosphorylation via CDK4/6 leads to the release of E2F transcription factors, which ultimately drive cell cycle progression 17,18,19,20
