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Zambrano AK, Cadena-Ullauri S, Ruiz-Pozo VA, Tamayo-Trujillo R, Paz-Cruz E, Guevara-Ramrez P, et al

Parkinsons disease MAO-B contributes to the pathogenesis of PD by boosting astrocytic GABA transmission and contributing to oxidative stress

Slc12a2 loss in insulin-secreting beta-cells links development of overweight and metabolic dysregulation to impaired satiation control of feeding

Concurrently, bioenergetic and dynamic rescue interventions seek to restore metabolic homeostasis using complex I chaperones, DRP1 inhibitors (for example Mdivi-1), and precision metabolic rescue like NDI1 gene therapy designed to entirely bypass the defective host complex I [95]

To address this, we assessed: 1