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Rather than slowing gastric emptying or mimicking incretin hormones, tesofensine directly modulates central nervous system neurochemistry to influence appetite, satiety signaling, and energy expenditure

[DOI] [PubMed] [Google Scholar] 53.Kim H, Jacobson EL, Jacobson MK

Key finding: AOD 9604 reduced visceral fat by 24% in GH receptor-deficient mice, confirming GH-R independent lipolytic mechanism

Intestinal epithelial claudins: expression and regulation in homeostasis and inflammation

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